Can my cat with hepatic lipidosis survive?
According to this study, 44 of the cats with fatty liver survived and 27 died.
Identifying the decisive causes of the difference between survival and death,
- It can be a basic data for determining the degree of treatment for veterinarians treating fatty liver,
- Cat owners will know the importance of paying close attention to these items during the checkup.
1.Ammonia: Ammonia produced by gut bacteria and somatic cells during protein breakdown is detoxified by the liver and excreted in the urine via the kidneys.
Branvine recognized that ammonia is critical to the survival of a cat's fatty liver, so we opted for a US patented milk thistle extract that facilitates the detoxification process.
In other words, Branvine Liver Alive was made from "Let's avoid cat death from fatty liver.".
2. Amylase: Amylase is a digestive enzyme that occurs in the pancreas and salivary glands, and its level is greatly increased in acute pancreatitis symptoms.
Since pancreatitis is a disease correlated with fatty liver, the condition of the pancreas is considered to be an important factor in determining survival.
Additional views of Branvine -> Lipase: As an enzyme secreted by the pancreas to break down fat, an increase in its level suggests a problem with the pancreas, so it is important to evaluate it with amylase.
3. Creatine Kinase: An enzyme found in the heart, brain, skeletal muscle, and other tissues that rises when muscle or heart cells are damaged.
Creatine kinase is implicated in heart function, suggesting that cats with hepatic lipidosis are more likely to have poor heart condition as well.
To summarize, the liver/pancreas/heart condition appears to determine the survival and death of cats due to fatty liver.
In addition to the veterinarian's story, go to the hospital and pay special attention to the results of the blood ammonia level, amylase, lipase, and creatine kinase.
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Hepatic Lipidosis in Cats (Fatty Liver Syndrome) Part 4
Branvine
